Cannon, Martha, and Squires, Richard (2008) Feline azotemia. In: Vetstream FELIS. Vetstream, pp. 1-3.
|PDF (Published Version) - Repository staff only - Requires a PDF viewer such as GSview, Xpdf or Adobe Acrobat Reader|
View at Publisher Website: http://www.vetstream.com/Felis/Content/D...
• Azotemia is an increased concentration of nonprotein nitrogenous compounds (eg urea and creatinine) in the blood. "Uremia" is the clinical syndrome that occurs as a result of severe azotemia due to abnormal renal function.
◦ Reduced glomerular filtration rate:
- Reduced renal perfusion (pre-renal azotemia).
- Primary renal disease (renal azotemia).
- Obstruction to urinary outflow (post-renal azotemia).
◦ Uroabdomen leading to reabsorption of urinary nitrogenous compounds (post-renal azotemia).
◦ Increased absorption of nonprotein nitrogenous compounds from the gastro-intestinal tract, eg GI bleeding, high protein meal.
◦ Increased protein catabolism, eg fever, trauma, burns, hyperthyroidism.
• A degree of pre-renal azotemia may be super-imposed on pre-existing renal or post-renal azotemia.
|Item Type:||Book Chapter (Reference)|
This is a Vetstream FELIS peer reviewed journal article that was first published online in November 2008. It is updated quarterly.
|FoR Codes:||07 AGRICULTURAL AND VETERINARY SCIENCES > 0707 Veterinary Sciences > 070706 Veterinary Medicine @ 100%|
|SEO Codes:||86 MANUFACTURING > 8609 Veterinary Pharmaceutical Products > 860902 Veterinary Diagnostics @ 50%|
86 MANUFACTURING > 8609 Veterinary Pharmaceutical Products > 860903 Veterinary Pharmaceutical Treatments (e.g. Antibiotics) @ 50%
|Deposited On:||27 Sep 2011 11:29|
|Last Modified:||27 Sep 2011 11:29|
Last 12 Months: 0
Repository Staff Only: item control page