Update on canine and feline parvoviral infections: who is infecting who?
Squires, Richard A. (2006) Update on canine and feline parvoviral infections: who is infecting who? Proceedings of the 49th Annual Congress of British Small Animal Veterinary Association. 49th Annual Congress of British Small Animal Veterinary Association , 20-23 April 2006, Birmingham, UK , pp. 1-4.
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In the northern hemisphere's summer of 1978, Canine parvovirus (CPV), the causative agent of canine parvoviral enteritis, appeared and spread rapidly all over the world. Serological surveys subsequently showed that dogs had not encountered CPV before 1974 in Europe. It is thought that CPV arose by mutation of a precursor parvovirus, probably one that had been circulating in Europe for some time among one or more carnivore species. An alternative and enticing suggestion was that CPV had arisen from a modified live feline panleucopenia vaccine virus; however this has not been supported by subsequent study. All CPV isolates clearly formed a single tight group, and were considered most likely to have been derived from a single, common ancestral virus. CPV is extremely closely related to feline panleucopenia virus (FPV) and has recently been described as a “host range variant of a feline virus”. Although remarkably similar to FPV, the 1978 strain of CPV specifically infected members of Canidae, whereas FPV was known to infect and cause disease in felids, procyonids (raccoons), mink (Mustela vison) and possibly foxes (Vulpes spp.), but not dogs.
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|Deposited On:||13 Oct 2011 17:22|
|Last Modified:||13 Oct 2011 17:25|
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